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MIT researchers have identified several promising new targets for Alzheimer’s disease treatment by integrating data from fruit flies, human brain tissue, and cell studies.
Their computational approach revealed previously overlooked cellular pathways that could contribute to neurodegeneration.
This discovery comes at a time when researchers are increasingly looking beyond the traditional amyloid plaque hypothesis to explain why the disease is so difficult to treat.
While still in early stages, the discovery of these alternative pathways offers hope for developing more effective treatments for Alzheimer's disease.
By addressing multiple causes simultaneously, future therapies might finally be able to significantly slow or even reverse the progression of Alzheimer’s disease.
The research, led by Matthew Leventhal , was funded by the National Institutes of Health .
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