Genetic Mutation Linked to Schizophrenia
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Mutation increases enzyme in mouse brains linked to schizophrenia behaviors

80% Informative
A genetic mutation found in two human patients with schizophrenia also increased schizophrenia-related behaviors in mice with the same mutation.
The mutation increases levels of glycine decarboxylase, an enzyme responsible for regulating glycine in the brain.
Glycine activates receptors for the neurotransmitter glutamate, called NDMA receptors.
Yet this finding also is relevant independently of disease, as the NMDA receptor is essential for many brain functions, including learning and memory.
This work was supported by the National Institutes of Health , the Shervert Frazier Research Institute at McLean Hospital , the Stanley Center for Psychiatric Research at the Broad Institute of Harvard and MIT .
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